receptors (Mann and Mody, 2010). It seems that ethanol may enhance GABAergic neurotransmission via increment of interneuron excitability and GABA release, and may decrease glutamatergic neurotransmission via reduction of glutamate release(Hendricson et al., 2004) and inhibition of NMDA receptor function(Lovinger et al., 1990), which causes striking imbalance between excitation and inhibition within neuronal network and impairment of γ oscillations (Figure 7).
