were removed. Furthermore, only SNPs which passed an exact test of Hardy–Weinberg equilibrium (p>5×10−7) were considered for analysis. Of these 8365 individuals, 5819 had BMI data, and 4251 had CRP and LDLc levels measured. In order to investigate the effect of specific SNPs from GWAS meta-analyses of BMI, CRP, and LDLc we used autosomal genotypic data that had been imputed using Markov Chain Haplotyping software (MACH v.1.0.16) and phased haplotype data from CEU individuals (Hapmap release 22, Phase II NCBI B36, dbSNP 126).
