of AUD and T1 typical quantity were positively related to the hazard function such that having an alcoholic parent and higher levels of baseline drinking were associated with AUDs across adulthood; and there was a negative relation between T1 age and AUD occurrence in the 20’s (significant in the 20–24 year range) such that an older age at the baseline interview was associated with a lower occurrence of AUD; this trend reversed in subsequent years with an older age at baseline interview becoming associated with a higher rate of AUD occurrence (significant in the 45–49 year range). The parameter estimates listed for the age intervals represent the negative logits of the hazard probability for each age interval and the corresponding percentages listed under odds ratios are the probability of AUD onset for those probands not considered missing at each age interval.
