is more strongly tied to psychological dependence, while drinking for social or sensation-seeking motives are associated with heavy/frequent use (Kuntsche et al., 2005), but despite different underlying mechanisms both could lead to AUD symptoms/diagnoses. If any one of these pathways is overrepresented in clinical samples (due perhaps to ascertainment bias from greater severity or medical/psychiatric comorbidities that increase treatment-seeking), the etiological overlap with AUD phenotypes in the general population would be reduced.
