conduction delays or even blockages in vivo, such as characteristically occurs in multiple sclerosis patients with optic neuritis (Cuypers et al., 1995), and c) schizophrenia patients have repeatedly been shown to exhibit white-matter abnormalities, both in vitro with microscopy (Uranova et al., 2007) and in vivo with structural MRI (Whitford et al., 2007) and, more recently, Diffusion-Tensor Imaging (DTI) (Kubicki et al., 2007). Thus there is reason to suspect that schizophrenia is associated with neural conduction delays caused by structural damage to white-matter fasciculi.
