The observed high SNP-based genetic correlation (consistent across two methods) suggests that largely the same common autosomal risk variants are involved in ADHD for both sexes. While sex-specific heterogeneity from common autosomal variants seems unlikely based on our results, we cannot rule out the possibility that heterogeneous effects exist for rare or nonautosomal variation or that with increased sample sizes, weaker effects of common variant heterogeneity could be detected.
