the liver’s total ethanol-oxidizing capacity; in addition, π-ADH accounts for nearly 30 percent (Hurley et al. 2002). The ethanol-oxidizing capacities of men with the same weight and BAC, but with different ADH1B and ADH1C alleles, also have been estimated (Lee et al. 2006). For a man homozygous for the ADH1B*1 and ADH1C*2 alleles, the total ethanol-oxidizing capacity is about 80 percent of the man homozygous for the reference alleles. For a man homozygous for the ADH1B*2 and ADH1C*1 alleles, the oxidizing capacity is almost eight times higher than that of the man homozygous for the reference alleles. For a man homozygous for the ADH1B*3 and ADH1C*1 alleles, the oxidizing capacity is almost twice that of the man homozygous for the reference alleles.
