the N2 (and the conflict-monitoring process it reflects) is differentially affected by alcohol as a function of trial-to-trial fluctuations in performance, however. In the current study, consistent with the idea that alcohol’s impairment of control can be characterized in terms of deficits in reinstating control following control failures, it was predicted that the CE in N2 amplitude would be attenuated by alcohol on post-error trials but not on trials following correct responses.
