Most importantly, these analyses revealed no significant group main effects, although there were three group × condition interactions (Table 3). For the temporal N1 sink, the marginal significance for this two-way interaction stemmed from greater amplitudes for targets than novels in controls (F[1,38] = 12.2, p = .001) but not patients (F[1,38] < 1.0). Also for factor 185, the greater mid-frontal sink for novels than targets (novelty MMN) was less robust for patients (F[1,38] = 9.66, p = .004) than controls (F[1,38] = 35.2, p < .0001), but there were no significant simple effects of group for targets (F[1,38] < 1.0) or novels (F[1,38] = 2.54, p = .12). For the response-related centroparietal source (factor 505), simple effects of condition were less robust for patients (F[1,38] = 5.10, p = .03) than controls (F[1,38] = 25.5, p < .0001). This group × condition interaction was further modified by a significant three-way interaction involving hemisphere, which originated from right-greater-than-left hemisphere differences in controls for novels (F[1,38] = 5.69, p = .02) but not targets (F[1,38] < 1.0), whereas the same asymmetry
