Mental disorders displayed similarly pronounced genetic overlap with intelligence, EDU, neuroticism, and SWB (fig. 3). As for mental disorders, AIC differences indicated that the MiXeR-modelled overlap was indistinguishable from maximum possible overlap for all analyses besides MD and NEUR, which displayed poor model fit (supplementary fig. 4, supplementary tables 6-7). Prominent mixed effect directions among shared ‘causal’ variants were supported by LAVA local correlations (supplementary results).
