2010), autism (unpublished, available via Psychiatric Genomics Consortium), bipolar disorder (Psychiatric GWAS Consortium Bipolar Disorder Working Group 2011), major depressive disorder (Major Depressive Disorder Working Group of the Psychiatric GWAS Consortium et al. 2013), schizophrenia (Schizophrenia Working Group of the Psychiatric Genomics Consortium 2014), anxiety disorders (Otowa et al. 2016), depressive symptoms, neuroticism, subjective well-being (Okbay, Baselmans, et al. 2016), anorexia (Boraska et al. 2014), body mass index (Locke et al. 2015), tobacco usage (Tobacco and Genetics Consortium 2010), reproductive behavior (Barban et al. 2016) and structural brain measures (accumbens, amygdala, pallidum, caudate, thalamus, putamen volumes) (Hibar et al. 2015), hippocampal volume (Hibar et al. 2017), intracranial volume (Adams et al. 2016), insomnia (Hammerschlag et al. 2017), educational attainment (Okbay, Beauchamp, et al. 2016), and alcohol consumption (Clarke et al. 2017; Schumann et al. 2016). We identified high confidence HapMap SNPs (for which the LD scores have been precomputed) present in the OCD summary statistics and each of the other summary statistics. For continuous traits (e.g. cognitive performance, brain structure volumes) no sample or population prevalence was specified. For binary traits the sample prevalence was calculated based on the reported number of cases in the sample, while the population prevalence
