the SORT1 and LPA lead SNPs, simply by being in weak LD with them. We also deferred studying FUT2, since its best eQTL SNP is a coding variant in the gene. Among the other top GLGC HLC eGenes, we sought to identify functional regulatory variants in the loci of CPNE1, ANGPTL3, and FRK, as described below. We note that in eQTL analyses performed only on higher-quality HLCs (the 63 HLC samples with scores of at least three in Table S1, out of the total 86 HLC samples), all of the top GLGC HLC eGenes remained strongly associated with SNPs in their loci (Figure 2B).
