thus appears reasonable to conclude that if the power in a spontaneous oscillatory frequency is high, there is a relatively higher probability that partial phase resetting of the frequency would render higher power to the phase-locked oscillatory frequency compared to a spontaneous oscillatory frequency that had lower power. However, there is evidence that points to lower phase locking rates among impulsive university students (Knyazev et al., 2008a), schizophrenia patients (Basar-Eroglu et al., 2009), and alcohol preferring rats (Criado and Ehlers, 2010). Further, the diffused brain dysfunction hypothesis that postulates that alcohol causes generalized deficits that involve many structures of the brain, is associated with an abnormal EEG profile, and dysfunction resembles that of premature aging (Glenn et al., 1994, Kamarajan, Porjesz, 2004, Newman, 1978, Parsons, 1994, Tivis et al., 1995) may also lend additional support to the present observation showing lower evoked power during task demands. In addition, structural magnetic resonance imaging (MRI) evidence of less white matter integrity and cellular volume loss in various parts of the brain in alcoholics may also help explain the relatively lower additive evoked power contributions, the lower rates of partial phase resetting during ERP generation, and therefore lower evoked power in various ERO
