Very recently it has been shown, that the priming of withdrawal reflexes in the context of chemosensory anxiety signals is intensified in non-clinical socially anxious participants [15]. Thereby, it is suggested that socially anxious people might process such signals with a stronger neuronal investment than non-socially anxious people. As it is generally agreed that social phobia is associated with a bias in the processing of social information [21], an intensified neuronal processing of social fear signals might be highly disorder-specific [22].
