for the “C” effect allele; I2=0) and p=0.0024 (B=0.054 for the “G” effect allele; I2=0), respectively). No genotyped proxy SNPs could be found for rs12461383 and rs1801272 SNPs. We did not replicate, with a nominal p<0.05, associations between SNPs in 7p14, 8p11 and 10q23 loci, reported in previous GWAS on CPD in our analysis on lifetime average CPD (online supplementary table 2).
