These findings highlight the importance of developing our understanding of the bidirectional relationship of impulsivity and alcohol both at the behavioral and biological level including gene expression and neurobehavioral functioning. Further, these findings show that both behavioral (i.e., delay discounting) and self-report measures of impulsivity can distinguish subtypes of individuals presenting with AUDs, and do so in a reasonably systematic manner. In addition to more straightforward relationships between impulsivity and AUDs, identifying these factors, such as psychopathology, violence/aggression, and biological mechanisms can serve to further elucidate the relationship between impulsivity and alcohol use. The limited research on psychopathology suggests that certain dimensions of impulsivity may manifest differently or to a greater extent for some individuals than others given issues of comorbidity. However, this literature is largely limited to focusing on Cluster B Personality Disorders, and thus more work is needed in examining how other types of PDs and Axis I disorders may also influence the relationship between impulsivity and AUDs. The reviewed studies also illustrated how co-occurring factors, such as violence and aggression, may shed light on aspects of the
