N2 are sensitive to different aspects of task processing, where lesion-induced attentional deficits led to impaired task processing (i.e., reduced ERN amplitude) while also causing increased processing of irrelevant stimulus information (i.e., increased N2 amplitude). Thus, findings are equivocal, and there is a dearth of studies evaluating the N2 component using a Go/NoGo task in alcoholics.
