to the base model, fits significantly better [F (1,3027) = 21.8, p = 3.2E−06] and accounted for an additional 0.66% of phenotypic variance in BMI for a total of 4.1%. We note that the GRSS accounted for more of the variance in BMI than either sex or age. Interactions between the covariates and the GRSS were tested but no significant interactions were found (presented in Supplemental Table 2). Therefore, our results suggest that GRSS was equally associated with BMI in men and women, in European- and African-Americans and across all ages.
