of stress and depression) and we investigated whether the hypothesized interaction could be more effectively detected using all available data with stress and depression assessed. In secondary analyses, we also examined multiple models for the coding of the genotype (additive, dominant, recessive, haplotypes) as well as broad and narrow requirements for documentation of temporal order of the stress experience and the onset of depression. Despite these efforts, we were unable to uncover specific subgroups where the G×E interaction was clearly expressed.
