a disinhibitory disorder [111] and may be related to neuronal hyperexcitability [108]. Interestingly, similar to the current findings, impulsivity factors ranked as the topmost predictors in our previous study on fMRI FC with the same set of participants [54]. Earlier studies have also drawn etiological connections among AUD, externalizing traits such as impulsivity, and neural disinhibition in the form of electrophysiological features in AUD individuals (e.g., low P3 amplitude and delta and theta oscillations underlying P3 during cognitive processing, and increased resting state beta power) [108,118,119,120]. Importantly, impulsivity was found to be associated with reduced P3 amplitude in AUD [120] and other externalizing disorders [157,158,159]. Further, recent studies have found an association of impulsivity with resting state measures of EEG power [160], EEG-based FC [161], and fMRI-based FC [3], suggesting that impulsivity in AUD may underlie specific brain networks.
