was observed [Δχ2(5)=4.44, p=0.49], so the reduced model was accepted. Next, we tested this reduced model against a model in which a single factor accounted for genetic influences on trauma exposure and PTSD and found no significant change in model fit [Δχ2(3)=0.16, p=0.92]. Variance in these three phenotypes was best represented with an ACE model for trauma exposure and AE models for PTSD and AD, with one genetic factor accounting for heritable influences on trauma exposure and PTSD and a second accounting for heritable influences on AD.
