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Chunk #43 — Results — Longitudinal genetic analyses

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Stable genetic effects on symptoms of alcohol abuse and dependence from adolescence into early adulthood.
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For unique environmental influences a simplex structure with innovation terms was specified, allowing for stable and dynamic influences. At younger ages the largest part of unique environmental influences was due to unique environmental innovation. According to the final model, at age 18–20, 98% of the unique environmental variance was due to innovation (.582/(.342 × .272 + .582) × 100%). At age 21–23, this was 83% (.502/[(.342 × .272 + .582) × .382 + .502] × 100%) and at age 24–26, 65% (.472/([(.342 × .272 + .582) × .382 + .502] × .632 + .472) × 100%). At later ages, the largest part of unique environmental influences consisted of those experienced earlier and transmitted to a later age. For example, at age 27–29, only 16% of unique environmental variance was due to innovation (.232/[([(.342 × .272 + .582) × .382 + .502] × .632 + .472) × .912 + .232] × 100%) and 84% to unique environmental factors experienced earlier in life. For age 27–29, 72% of unique environmental influences consisted of those transmitted from earlier ages and 28% was due