inputs as well as glutamatergic synapses arising from thalamus (LeDoux et al., 1991), hippocampus (Kishi et al., 2006), limbic and parietal cortex (McDonald and Mascagni, 1996), and intra-amygdala projections (Savander et al., 1995). The contrasting results found in the current study and in the Lack et al. 2007 study suggest that the neural circuitry contributing to distinct glutamatergic release pathways in the BLA might be differentially sensitive to chronic ethanol exposure and withdrawal.
