weeks between sessions. LTAA were more prone to social deviance as evidenced by lower CPI-sociability scores (17 vs. 22, η2 = 29.7, p < 0.0001), were more impulsive (9 vs. 3, η2 = 37.3, p < 0.0001), and had greater antisocial disposition (23 vs. 16, η2 = 31.9, p < 0.0001). LTAA also had higher ASPD lifetime symptoms counts (7.6 vs. 3.0, η2 = 25.6, p = 0.001) but comparable current ASPD symptom counts (1.25 vs. 0.67, η2 = 4.2, NS). These results imply that LTAA were able to inhibit their antisocial behavior despite an unchanged antisocial disposition.Table 1Demographics, alcohol/drug use measures, time between EEG and fMRI acquisition, and psychological measures of Non-Substance Abusing Control (NSAC) and Long-Term Abstinent Alcoholic (LTAA) participants.Table 1.Characteristic/measureNSAC (n = 21)LTAA (n = 20)Effect size partial η2(% variance)Odds ratioMean or nSD or %Mean or nSD or %DemographicsAge (yrs)47.336.6447.206.950.00Education (yrs)15.482.1613.852.0313.6⁎Female, n (%)733.33%735%1.08Weeks between EEG and fMRI6.578.244.659.481.2Alcohol/drug useLifetime average drinks/month10.768.53189.07165.3638.5⁎⁎, aPeak use drinks/month17.8615.04334.10263.1543.7⁎⁎, aLength of abstinence (days)––2749.352933.62–Lifetime nicotine Use, n (%)314.29%1260%9⁎Cannabis use, n (%)2b9.52%2b10%1.06Cocaine use, n (%)1b4.76%00%0Psychological measuresMMPI psychopathic deviance scale6.578.244.659.4831.9⁎⁎⁎Eysenck impulsivity scale3.382.789.054.5837.3⁎⁎⁎CPI socialization scale22.243.8317.104.2829.7⁎⁎⁎ASPD lifetime symptoms3.003.077.604.8225.6⁎⁎ASPD current symptoms0.670.971.251.804.2⁎p < 0.05.⁎⁎p < 0.001.⁎⁎⁎p < 0.0001.aStatistical comparisons are inappropriate since the variable is related to selection criteria.bRecreational substance
