A growing number of studies have examined fMRI based resting state functional connectivity in AUD and found aberrations in neural communications across brain regions in several networks (e.g., [23,26,28,29]). Specific to DMN, Chanraud et al. [21] were the first to report compromised functional connectivity across the DMN nodes, showing that the posterior cingulate and cerebellar regions in alcoholics had lower synchronization (hypo-connectivity) compared to controls. Muller-Oehring et al. [26] reported that alcoholics showed DMN related aberrations of fMRI hypo-connectivity of posterior cingulate with right caudate region and hyper-connectivity of PCC with right occipital regions, further implicating the posterior cingulate region. In contrast, Kim et al. [28] reported that AUD individuals did not differ in fMRI FC of the DMN but manifested lower FC in the executive control network and the dorsal attention network. Zhu et al. [3] investigated multiple regions involving several networks and found that individuals with AUD exhibited within-network hyperconnectivity in DMN, salience, orbitofrontal cortex, left executive control and amygdala-striatum networks. Recently, in another fMRI FC study using random forest (RF), Zhu et al. [29] reported that executive
