of alcohol use/abuse on the brain, and that this effect at least partially resolves with extended abstinence. We hypothesized that (1) evoked theta power would be reduced to the same degree in both STAA and LTAA compared with NAC, given evoked theta's role as a putative marker for the genetic vulnerability to alcoholism (which should not differ as a function of length of abstinence), but that (2) like LTAA, the STAA group would show increased induced theta ERS, but to a greater degree, that is, the magnitude of the theta ERS would be greater in STAA (given their recent initiation of abstinence from alcohol) compared with both LTAA (who have orders of magnitude longer abstinence than STAA) and controls.
