Previous research has shown that individuals who exhibit larger ERNs on error trials tend to display better response control, as reflected in PDP control estimates (e.g., Amodio et al., 2004; Amodio, Devine, & Harmon-Jones, 2008). Such an association is consistent with the view that the ERN reflects an evaluative signal to other cortical structures that more regulative control is needed (Botvinick et al., 2001; Botvinick, Cohen, & Carter, 2004; Kerns et al., 2004). Given that alcohol diminishes this evaluative control signal, the typical association between the ERN and response control also is likely to be disrupted. To test this idea, we computed correlations between ERN amplitudes and PDP control estimates, separately by beverage group (see Figure 6). For participants who did not consume alcohol (i.e., placebo and control groups), the typical association was observed—larger ERNs were associated with increased PDP control (rs ≥ −.47, ps < .05). This association was absent in the alcohol group, however (r = −.09, p > .30). A multiple regression model in which PDP control scores were regressed on beverage group (coded 0 = alcohol,
