The purpose of this study was to further investigate the interaction between ethanol intake and dietary fat and determine if TG and hypothalamic peptides may contribute to this relationship. To this end, four experiments were conducted which examined: 1) the interaction between fat and ethanol; 2) the association of fat and ethanol with TG levels; 3) a possible pharmacological means for reversing this relationship; and 4) a neural mechanism underlying this reversal.
