At the same time, it is well known that the liability for neurological and psychiatric disorders is under genetic control (Polderman et al., 2015). In the beginning of this century, many argued that investigating EEG as an intermediate phenotype (or endophenotype) would aid in finding specific genes for behavioral traits and mental health disorders (de Geus, 2010). This idea was surpassed by the massive case–control genome‐wide association studies (GWAS) performed in human genetics (Bulik‐Sullivan et al., 2015; Sullivan 2010). However, the black box method of GWAS leaves unexplained how specific risk variants exert their influence on the brain on a systems level (de Geus, 2010). This is the main focus of our consortium: to find how genetic variants influence individual variation in EEG phenotypes and to link these to variants affecting brain disorders.
