Lastly, we parsed each disease variant position for overlap with iPSC eQTLs, again considering lead variants and their high-LD proxies for each tissue as follows. We report all disease variants that were tagged by iPSC eQTLs (lead or proxy) and the eGene(s) regulated by the eQTL lead/proxy (Supplementary Table 6a). We additionally report a subset of these variants that are exact matches with iPSC lead eQTL variants and highlight the number of high-LD proxies (r2 > 0.8) each variant has (Supplementary Table 6b).
