Several studies have reported structural temporal lobe abnormalities in schizophrenia, including superior temporal gyrus and hippocampus (e.g., Arnold et al., 1991; Shenton et al., 1992; Bogerts et al., 1990, 1993; Menon et al., 1995; Pearlson et al., 1997), brain structures critical involved in the formation and retrieval of memory representations (e.g., Damasio, 1989; Smith and Halgren, 1989). Neuroimaging studies have provided evidence linking the verbal memory deficits in schizophrenia to left medial temporal lobe (MTL) structures (e.g., Gur et al., 1994; Mozley et al., 1996; Nestor et al., 2007). It has been suggested that structural abnormalities of the temporal lobe are directly linked to the frequently-reported reductions of event-related brain potentials (ERPs) in schizophrenia, most notably of the late cognitive P3 component (e.g., McCarley et al., 1991, 1993, 2002, 2008; O’Donnell et al., 1993, 1999; Egan et al., 1994; Kawasaki et al., 1997). Although several studies supported the concept that both structural (e.g., Barta et al., 1990; Rossi et al., 1992; Bilder et al., 1994; Faux et al., 1993; Falkai et al., 1995; Vita et al., 1995) and functional abnormalities
