Mean CSD waveforms and ERSP plots for the three converters are shown at selected sites in Figure 8. Due to the extremely small subsample size, CSD waveforms were more volatile but were nevertheless characterized by robust condition effects, which included a distinct NVS for novels at Cz and a late P3b for targets with a Pz maximum (Fig. 8A). However, in striking contrast to controls and the overall sample of patients, the novelty MMN, which partially overlaps but follows N1 sink and uniquely distinguishes novel stimuli from targets (cf. Fig. 3, row 2), was not observed for these individuals. To highlight this observation, a shaded area was added to Fig. 8A depicting the difference from controls for novel stimuli. Fig. 9A compares novel CSDs for controls and CHR patients with and without transition to psychosis at sites where novelty MMN and NVS were most robust (i.e., at FCz and Cz; cf. Fig. 4), and Fig. 9B depicts novel-minus-nontarget difference waveforms at FCz where novelty MMN (factor 185) was maximal so as to mimic deviant-minus-standard MMN waveforms computed in passive auditory
