The key hypotheses and aims of the project are: Hypothesis 1: Participants with mood disorders have poorer neuropsychological function in the cognitive dimensions of cognitive function, emotion processing, and social cognition compared to healthy matched controls.Hypothesis 2: Participants with mood disorders have poorer quality of life and poorer psychosocial functional outcomes that will correlate with the level of impairment in cognitive dimensions of depression.Hypothesis 3: Participants with mood disorders show a biological genomic “signature” that relates to cognitive dimensions of depression.
