CeA and medial amygdala, but not the basolateral amygdala (Pandey et al. 2005). Further, virally mediated alterations in NPY levels in the CeA differentially affect EtOH consumption in rats with low and high basal levels of anxiety (Primeaux et al. 2006). Also, recent genetic and pharmacological evidence indicates that C57BL/6 J mice have low NPY levels in CeA compared to DBA/2 mice, suggesting that NPY contributes to the high EtOH consumption characteristic of C57BL/6 J mice (Hayes et al. 2005).
