The data reported here provide evidence of reduced ITPC in the α-band in ASD. Together with the result of Rojas et al. (2008), these data indicate widespread dysfunction of neural timing in ASD, rather than a specific deficit of high-frequency γ-band oscillations as some authors have predicted. Reduced ITPC in ASD indicates that those with ASD are less able to synchronize the activity of stimulus-related cell assembly(ies) consistently across trials, and provide evidence for temporal disruptions in the organization and recruitment of cell assemblies. It is not clear whether this temporal disruption underpins, is caused by, or is unrelated to, postulated neural de-synchrony in ASD.
