In summary, in keeping with similarly sized previous genome-wide association studies of ADHD, we failed to find significantly associated common variants. We previously found large, rare CNVs to be associated with ADHD, and the results remain similar in this newly extended sample. Contrary to what some might expect, we found a highly significant overlap of biological pathways hit by both CNVs and SNPS. This implies that both types of gene variants are relevant to ADHD risk. Finally, our results suggest that CHRNA7 is a promising candidate to examine further.
