Therefore, the first two factors clearly corresponded to the alpha ERD (factor 610–9) and the preceding ERS coincided with the NVS (factor 260–1). This interpretation was corroborated by the condition-dependent topographies, which revealed 1) a prominent ERD for target stimuli over centroparietal sites, with a right hemisphere maximum that was greater for controls than patients (Fig. 7B, alpha ERD, column 2), and 2) a robust ERS for novel stimuli over mid-frontocentral sites (Fig. 7B, NVS ERS, column 3). Likewise, factor 130–1 clearly corresponds to the early ERS coinciding with the N1 sink, revealing topographies that implicate synchronized activities at the defining sink/source maxima of N1 over each hemisphere (i.e., C3-TP9 and C4-TP10) for nontarget and target stimuli, and at locations with maximum temporal N1 sink activity (e.g., T8) for novel stimuli (Fig. 7B, N1 ERS; cf. also Fig. 4B, N1). In addition to the close temporal conjunction between the tPCA and tfPCA factors associated with N1 sink and NVS, the bilateral N1 sink/source dipole pairs of grand mean CSD waveforms for targets corresponded to half-cycle sinusoids of 3.7 Hz, and
