execution mentioned above, whereas this low frequency synchronization in NoGo trials is apparently needed to suppress undesirable response. This assumption is in accordance with ERP studies showing relation of the P3-NoGo component and anteriorization of this component to response inhibition, rather than to conflict monitoring [35], [40], [41], [42]. Higher frontal theta synchronization in NoGo trials could be a modulation frequency for the frontal ERP component N2, which is strongly enhanced in NoGo trials and diminished or even absent in Go trials [7], [43], and assumedly reflects a more general process of conflict monitoring, rather than suppression of a motor response [7], [34], [43]. Like N2 ERP component, the source of theta oscillatory activity in this case is localized in or received modulator input from ACC, which is mostly activated by conflict monitoring and response competition [4], [5], [37].
