Consistent with our hypothesis, we found evidence for two forms of gene-environment interplay involving the role of social support: rGE and GxE. Supporting the notion that rGE as an important pathway linking genetic risk to developmental outcomes, we found that alc-GPS was associated with lower levels of family support, which in turn was associated with greater levels of alcohol use. This finding of gene-environment correlation can reflect evocative (or reactive) rGE whereby individuals’ predispositions and related traits influence how others respond to them (e.g., the degree of support they provide). Further, since our sample included adults spanning multiple developmental stages, it is also possible that this gene-environment correlation reflects active rGE processes as well such that individuals’ alc-GPS and related traits contribute to their choices and selections of environments (e.g., family). Generally, our finding is consistent with prior research demonstrating that rGE in the family context as an important pathway of risk to alcohol use outcomes. For example, Elam et al. (2018) showed that polygenic risk for aggression was associated with lower levels of family cohesion in adolescence, which in
