More recent studies have provided further evidence for genetic influences on P3 and addressed a number of related questions, including genetic covariation between P3 latency measured in a delayed response working memory task and working memory performance (Hansell et al., 2005), genetic specificity of P3 as evidenced by the lack of genetic overlap with other ERP measures such as MMN or P50 (Hall et al., 2006a), invariance of P3 heritability across the lifespan demonstrated using cross-sectional comparison of young and middle-aged adult cohorts (Smit et al., 2007a), genetic influences on age-related decrease of P3 amplitude using longitudinal analysis spanning ages from 17 to 23 (Carlson and Iacono, 2006) and genetic influences on neural oscillations contributing to P3 assessed using time-frequency decomposition of event-related EEG (Ethridge et al., 2012).
