Models 2 and 3 differed in the explained amount of variance in obesity: 12% versus 18%, respectively. As illustrated in Figure 2, the two models also differed in the route by which the construct of mood dysregulation (i.e., the variance shared by depression symptoms, conduct problems, and stimulant dependence symptoms) was linked to obesity. In Model 2, the path was direct, but not statistically significant (β=.14, p=.38). In Model 3, the path was indirect and significant: The effects of mood dysregulation were mediated (β= −.16, p=.05) through anterior CNVamplitude, which was, in turn, related (β= −.26, p<.001) to obesity.
