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Chunk #82 — Studies of EEG Biofeedback in Substance Abuse Treatment — Continuing Research — Self-Perception and Experimental Schemata in the Addicted Brain

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EEG biofeedback as a treatment for substance use disorders: review, rating of efficacy, and recommendations for further research.
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Research indicates that substance abusers have elevated beta activity in an EEG resting state as compared with normative groups (Rangaswany et al. 2002) and elevated alpha activity after administering a mood altering substance (Cohen et al. 1993; Kaplan et al. 1985). It is suggested that many of the neurophysiological markers may provide information about the state of the individual prior to the development of an addictive disorder and that these brain functions are under genetic control (Porjesz et al. 2002, 2005; Tapert 2004). Kaplan et al. (1985) reported lower frontal alpha and slow-beta coherence in alcohol-dependent males and females. Michael et al. (1993) found higher central alpha and slow-beta coherence, but lower parietal alpha and slow-beta coherence in males with alcohol dependence; contrarily, other findings suggest that morphine, alcohol and marijuana show increased alpha 2 power in the spectral EEG and relate this to the euphoric state produced by the drug (Lukas 1989, 1995). Winterer et al. (2003a, b) described higher left-temporal alpha and slow-beta coherence and higher slow-beta coherence at right-temporal and frontal electrode pairs in alcohol-dependent males and