are different in the examined autistic subjects than those in tuberous sclerosis heterotopias or focal cortical dysplasia. The development of the giant neuron- and balloon cell-free dysplasias observed in the autistic subjects might be related to differences in cause and/or mechanism. The detection of changes similar to focal cortical dysplasia in association with prenatal ischemia [65] or in shaken infant syndrome [74] may support these speculations.
