Most studies on the links between binge drinking and resting-state functional connectivity have focused on the reward, salience, and executive control networks. The most replicated finding is the negative association between alcohol misuse and connectivity within the reward network, specifically, between amygdala and orbitofrontal cortex (OFC; Crane et al., 2018; Peters et al., 2017, 2015). On the other hand, when assessing different seeds of the reward network (i.e., nucleus accumbens (NAcc)-OFC, instead of amygdala-OFC), an opposite pattern was reported such that compared to young adult light drinkers, binge drinkers showed increased NAcc-OFC connectivity (Arienzo et al., 2019). Functional connectivity between the salience and other networks has also been associated with alcohol misuse, with salience-reward (dorsal anterior cingulate cortex (dACC)-amygdala) connectivity associating negatively with alcohol misuse in a group of non-dependent alcohol drinkers (Hu et al., 2018), and binge drinkers showing increased salience-habit network (dACC-caudate) relative to light drinkers (Arienzo et al., 2019). Regarding the executive control network, both increased (Sousa et al., 2019) and decreased (Weiland et al., 2014) connectivity within this network were reported in binge drinkers compared to
