show evidence for association, this cannot be interpreted as failure to replicate previous results in all cases, because much of the variation reported as associated is not captured on the Affymetrix platform (e.g. length polymorphisms, microsatellites, untagged SNPs such as the promoter variant at MET21). Instead, despite a high density of markers, our results suggest that we did not identify additional common variation with evidence for association. Overall however, our results imply that these postulated candidate regions, mostly based on rare events known to cause autism, are not among the regions with common alleles having the strongest risk effects for autism.
