The effects of chronic alcohol intake amount on subsequent normal CNS neuroelectric activity have been little studied, although there are intriguing hints. High-alcohol drinking (HAD) rats demonstrated greater spectral power for the delta (2–6 Hz) and theta (4–6 Hz) bands compared to low-alcohol drinking (LAD) rats, with both groups specifically bred for their drinking proclivity. Further, HAD rats exhibited increased activity in the fast beta (13–30 Hz) and high-voltage fast alpha (9–12 Hz) bands [30]. These findings are consistent with EEG effects observed in binge drinkers who had not consumed alcohol at the frequency that is normally associated with alcoholism.
