Nonalcoholic control subjects exhibited larger late P3 components to target stimuli compared with nontarget stimuli (figure 1a). Alcoholics, however, manifested reduced or absent P3 components that differed little in amplitude, whether the stimulus was a target or a nontarget (figure 1b) (for review, see Porjesz and Begleiter 1983, 1985, and 1993).
