which is flanked by two segmental duplications. However, as with all CNV analyses of single-nucleotide polymorphism (SNP) array data, our study had limited resolution to establish the nature of potentially complex rearrangements at this locus; therefore, we cannot exclude the possibility that some of the duplications identified at 15q13 are of a more complex nature. The presence of the 15q13.3 duplication also seemed to modulate the ADHD phenotype, as carriers had a higher lifetime rate of comorbid conduct disorder.
