Binge drinking is common 1 and associated with significant health risks (e.g., previous studies 2 , 3 , 4 , 5 ). The impact on risk of how one consumes alcohol (how quickly and how high an alcohol concentration is achieved) is inherent in the definition of binge and high‐intensity drinking. The National Institute on Alcohol Abuse and Alcoholism (NIAAA) explicitly recognizes binging (“… a pattern of drinking that brings blood alcohol concentration (BAC) levels to [80 mg/dL]”) as one pattern of risky drinking, typically occurring after four or five drinks for women and men—in about 2 h. 6 Breath alcohol concentration (BrAC) indexes the arterial concentration (as in Lindberg et al. 7 ), to which the brain is exposed. 8 Unfortunately, many individuals consume more than four or five alcohol drinks on an occasion. This pattern, termed high‐intensity drinking, 9 is associated with an elevated risk of developing an alcohol use disorder (AUD). 10 , 11 , 12 , 13 Binging and high‐intensity drinking are also clearly influenced by genetic risk; existing and novel risk loci were associated with
