structures, due to chronic alcohol exposure [4,15,16,17]. Recent studies have identified that neurostructural damage in AUD was significantly related to functional deficits in executive performance, which was in turn associated with microstructural changes in large-scale brain networks [18,19]. In summarizing brain changes due to chronic alcohol addiction, Sullivan and Pfefferbaum [20] stated that (i) alcoholism affects selective brain systems and circuits, and (ii) alcoholism causes “incomplete brain lesions” without destroying the neurons, leaving the potential for recovery. These findings and insights from the literature suggest the importance of understanding brain networks affected in AUD, and there is a fast-growing number of studies that attempt to elucidate network mechanism and dysfunction in AUD (e.g., [21,22,23,24,25,26,27,28,29,30]).
